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Additionally, mRNA expression levels of RUNX-2 and ALP, two key osteogenic markers, were found to be 1.6- and 1.8-fold higher, respectively, in differentiated cells compared to undifferentiated controls

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Both cell subsets were involved in implementing the onco-fetal features of the TME by interacting via Notch and VEGF signaling and contributing to the formation of an immunosuppressive environment (72)